Detalhe da pesquisa
1.
APOL1 risk variants in individuals of African genetic ancestry drive endothelial cell defects that exacerbate sepsis.
Immunity
; 54(11): 2632-2649.e6, 2021 11 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-34715018
2.
Expression of active B-Raf proto-oncogene in kidney collecting ducts induces cyst formation in normal mice and accelerates cyst growth in mice with polycystic kidney disease.
Kidney Int
; 102(5): 1103-1114, 2022 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-35760151
3.
Overexpression of TGF-ß1 induces renal fibrosis and accelerates the decline in kidney function in polycystic kidney disease.
Am J Physiol Renal Physiol
; 319(6): F1135-F1148, 2020 12 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-33166182
4.
Periostin overexpression in collecting ducts accelerates renal cyst growth and fibrosis in polycystic kidney disease.
Am J Physiol Renal Physiol
; 315(6): F1695-F1707, 2018 12 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-30332313
5.
Integrin-Linked Kinase Signaling Promotes Cyst Growth and Fibrosis in Polycystic Kidney Disease.
J Am Soc Nephrol
; 28(9): 2708-2719, 2017 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-28522687
6.
Phosphodiesterase Isoform Regulation of Cell Proliferation and Fluid Secretion in Autosomal Dominant Polycystic Kidney Disease.
J Am Soc Nephrol
; 27(4): 1124-34, 2016 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-26289612
7.
Periostin promotes renal cyst growth and interstitial fibrosis in polycystic kidney disease.
Kidney Int
; 85(4): 845-54, 2014 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-24284511
8.
The key role of NLRP3 and STING in APOL1-associated podocytopathy.
J Clin Invest
; 131(20)2021 10 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-34651582
9.
Going from acute to chronic kidney injury with FoxO3.
J Clin Invest
; 129(6): 2192-2194, 2019 05 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-31063992
10.
Antioxidant effects of ethyl acetate extract of Desmodium gangeticum root on myocardial ischemia reperfusion injury in rat hearts.
Chin Med
; 5: 3, 2010 Jan 22.
Artigo
em Inglês
| MEDLINE | ID: mdl-20180993